PhD in hemato-oncology
PhD in hemato-oncology
  • Utrecht, Nederland
  • 27 november 2024

PhD candidate focusing on improving allogeneic hematopoietic cell transplantation and chimeric antigen receptor (CAR) T cell therapy in pediatric patients with acute leukemia by looking at clinical parameters, tumor-intrinsic features and the functional dynamics of the immune system. In this project, I combine wet-lab assays with big data analysis in the programming language R. I look forward to continuing doing research in the field of translational immuno-oncology, thereby expanding my knowledge and contributing to the field and patient outcomes. I am also highly passionate about sex- and gender-based medicine, which I would like to implement in my future career. I would describe myself as ambitious, eager to learn, enthusiastic, and accountable.

Categorie
E-mail
lindedekker@hotmail.com

Opleiding

MASTER OF SCIENCE IN CANCER & STEM CELLS, UTRECHT UNIVERISTY
sep 2017 — nov 2019
MINOR IN ENGLISH, VRIJE UNIVERSITEIT AMSTERDAM
sep 2016 — jan 2017
EXCHANGE SEMESTER AT LUND UNIVERSITY, SWEDEN
aug 2015 — jan 2016
BACHELOR OF SCIENCE IN BIOMEDICAL SCIENCES, UTRECHT UNIVERSITY
sep 2013 — mei 2017

Ervaring

PHD CANDIDATE, UTRECHT UNIVERSITY, NIERKENS LAB
jan 2020 — sep 2024

With immunological assays and flow cytometry, we use in vitro models and patient material to study outcome predictors and potential improvements for allogeneic hematopoietic cell transplantation and CAR T cell therapy in pediatric patients with high-risk hematological malignancies.

RESEARCH INTERN, HARVARD UNIVERSITY, CAMARGO LAB
jan 2020 — sep 2019

Using cell culture, western blot, qPCR and mouse xenograft models, the sensitivity of novel Hippo pathway inhibitors targeting YAP in hepatocarcinoma was studied.Off target effects of these small molecule inhibitors were tested and ‘on target’ engagement was validated.

RESEARCH INTERN, UTRECHT UNIVERSITY, KUIPER LAB
okt 2017 — sep 2018

1) With the smMIP approach and subsequent sequencing, we characterized germline mutations in genes linked to asparaginase enzyme activity and host toxicity in pediatric acute lymphoblastic leukemia.
2) With PCR, qPCR and Sanger sequencing, we determined the relevance of subclonal IKZF1 deletions encompassing exon 4 to 7 for relapse development in pediatric acute lymphoblastic leukemia

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